The Role of Inflammation in Atrial Fibrillation following Open Heart Surgery

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dc.authoridHuseyin, Serhat/0000-0003-4118-040X
dc.contributor.authorCanbaz, S.
dc.contributor.authorErbas, H.
dc.contributor.authorHuseyin, S.
dc.contributor.authorDuran, E.
dc.date.accessioned2024-06-12T10:52:48Z
dc.date.available2024-06-12T10:52:48Z
dc.date.issued2008
dc.departmentTrakya Üniversitesien_US
dc.description.abstractThis study investigated the role of systemic inflammation in the development of atrial fibrillation (AF) after coronary artery bypass grafting (CABG). CABG was performed using cardiopulmonary bypass in 77 patients. Pre-operative AF was present in six patients (7.8%) and postoperative AF developed in 13 (18.3%) of the 71 patients with pre-operative sinus rhythm. Post-operative mediastinal drainage was significantly increased in patients with post-operative AF compared with those with sinus rhythm. Plasma E-selectin, P-selectin and vascular cell adhesion molecule levels were not significantly different between patients with pre- and post-operative sinus rhythm, those with pre-operative sinus rhythm and post-operative AF, and those with pre- and post-operative AF. There were significant differences between pre- and post-operative C-reactive protein, interleukin (IL)-6 and IL-10 levels within all three groups, but no differences in these parameters between the groups. Thus, in all groups there were significant alterations in mediators indicative of systemic inflammation following CABG, but comparisons between the groups revealed no differences predictive of AF.en_US
dc.description.sponsorshipTrakya University [TUBAP-515]en_US
dc.description.sponsorshipThis study was supported by the Trakya University Research Fund (TUBAP-515; S Canbaz principal investigator).en_US
dc.identifier.doi10.1177/147323000803600526
dc.identifier.endpage1076en_US
dc.identifier.issn0300-0605
dc.identifier.issn1473-2300
dc.identifier.issue5en_US
dc.identifier.pmid18831903en_US
dc.identifier.scopus2-s2.0-54449086535en_US
dc.identifier.scopusqualityQ3en_US
dc.identifier.startpage1070en_US
dc.identifier.urihttps://doi.org/10.1177/147323000803600526
dc.identifier.urihttps://hdl.handle.net/20.500.14551/18842
dc.identifier.volume36en_US
dc.identifier.wosWOS:000260085000026en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherSage Publications Ltden_US
dc.relation.ispartofJournal Of International Medical Researchen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectATRIAL FIBRILLATIONen_US
dc.subjectCORONARY ARTERY BYPASS GRAFTINGen_US
dc.subjectCARDIOPULMONARY BYPASSen_US
dc.subjectINFLAMMATIONen_US
dc.subjectE-SELECTINen_US
dc.subjectP-SELECTINen_US
dc.subjectC-REACTIVE PROTEINen_US
dc.subjectVASCULAR CELL ADHESION MOLECULEen_US
dc.subjectINTERLEUKIN-6en_US
dc.subjectINTERLEUKIN-10en_US
dc.subjectC-Reactive Proteinen_US
dc.subjectAdhesion Molecule Expressionen_US
dc.subjectCardiopulmonary Bypassen_US
dc.subjectE-Selectinen_US
dc.subjectMechanismsen_US
dc.subjectArrhythmiasen_US
dc.subjectMarkersen_US
dc.subjectVcam-1en_US
dc.subjectModelen_US
dc.titleThe Role of Inflammation in Atrial Fibrillation following Open Heart Surgeryen_US
dc.typeArticleen_US

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