The importance of pulmonary artery circulation during cardiopulmonary bypass

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dc.authoridCIKIRIKCIOGLU, MUSTAFA/0000-0003-1143-347X
dc.authorwosidCanbaz, Sevgi/AAD-5192-2020
dc.contributor.authorEge, T
dc.contributor.authorCanbaz, S
dc.contributor.authorCikirikcioglu, M
dc.contributor.authorArar, C
dc.contributor.authorEdis, M
dc.contributor.authorDuran, E
dc.date.accessioned2024-06-12T11:13:07Z
dc.date.available2024-06-12T11:13:07Z
dc.date.issued2003
dc.departmentTrakya Üniversitesien_US
dc.description.abstractThis study sought to determine changes in transpulmonary difference in blood cells and alveolar-arterial oxygen (A-aO(2)) gradient when pulmonary artery circulation was obstructed in patients undergoing coronary artery bypass grafting (CABG) with cardiopulmonary bypass (CPB). Eighteen patients were divided into group A (control group; X-clamp placed on aorta, n = 9) and group B (pulmonary ischaemia group; X-clamp placed on aorta and pulmonary artery, n = 9). Haematological parameters were compared before CPB and up to 90 min after declamping. A-aO(2) gradient differences were compared before and 2 h and 6 h after declamping. A transpulmonary increase in leucocyte levels normalized after 60 min in group A but remained higher in group B. A transpulmonary increase in neutrophils normalized after 60 min in group A and 90 min in group B. Increased lymphocyte levels normalized after 30 min in group A and 90 min in group B. A-aO(2), gradient was determined as: group A (294.8 +/- 74.3) and group B (321.2 +/- 73.3) before X-clamping; group A (132.7 +/- 22.7) and group B (236.6 +/- 41.5) 2 h after declamping; and group A (72.2 +/- 22.7) and group B (189.4 +/- 88.9) 6 h after declamping. When pulmonary artery circulation was obstructed during the X-clamping period, leucocyte, neutrophil and lymphocyte sequestration within both lungs increased, and an increased A-aO(2) gradient was observed because of tissue damage. To prevent post-operative complications, precautions to maintain normal pulmonary artery circulation are recommended.en_US
dc.identifier.doi10.1177/147323000303100103
dc.identifier.endpage25en_US
dc.identifier.issn0300-0605
dc.identifier.issue1en_US
dc.identifier.pmid12635529en_US
dc.identifier.scopus2-s2.0-0037228620en_US
dc.identifier.scopusqualityQ3en_US
dc.identifier.startpage17en_US
dc.identifier.urihttps://doi.org/10.1177/147323000303100103
dc.identifier.urihttps://hdl.handle.net/20.500.14551/23427
dc.identifier.volume31en_US
dc.identifier.wosWOS:000181291300003en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherCambridge Med Publen_US
dc.relation.ispartofJournal Of International Medical Researchen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectPulmonary Artery Circulationen_US
dc.subjectPulmonary Ischaemiaen_US
dc.subjectPulmonary Leucocyte Sequestrationen_US
dc.subjectCardiopulmonary Bypassen_US
dc.subjectX-Clampen_US
dc.subjectLung Injuryen_US
dc.subjectIschemia-Reperfusionen_US
dc.subjectPerfusionen_US
dc.subjectSurgeryen_US
dc.titleThe importance of pulmonary artery circulation during cardiopulmonary bypassen_US
dc.typeArticleen_US

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