Retrospective evaluation of the cases prediagnosed as viral encephalitis in Trace University Hospital between the period of 2000-2005

dc.contributor.authorYulugkural, Zerrin
dc.contributor.authorCelik, Ayguel Dagan
dc.contributor.authorCelik, Yahya
dc.contributor.authorKuloglu, Figen
dc.contributor.authorBueyuekkoyuncu, Nlluefer
dc.contributor.authorTansel, Oezlem
dc.contributor.authorAkata, Filiz
dc.date.accessioned2024-06-12T11:00:23Z
dc.date.available2024-06-12T11:00:23Z
dc.date.issued2008
dc.departmentTrakya Üniversitesien_US
dc.description.abstractIn this study, a total of 17 adult patients (( :18 years old; 12 male, 5 female) with encephalitis followed up in neurology and infectious diseases clinics of Trace University Hospital between the years 2000-2005 were retrospectively analyzed. The most common signs and symptoms were confusion (n: 13; 76.4%), nausea and vomiting (n: 13; 76.4%), disorientation (n: 12; 70%), fever and headache (n: 11; 64.7%), amnesia (n: 10; 58.8%), convulsions (n: 9; 52.9%), agitation (n: 7; 41%), dysphasia and aphasia (n: 6; 35.2%), nuchal stiffness (n: 5; 29.4) and focal neurological signs (n: 1; 5.8%). Six of the patients were admitted to the hospital during summer, six during winter, four during spring and one during autumn. Eleven (64.7%) of the patients had electroencephalographic signs compatible with encephalitis. Encephalitis related signs were detected in 83.3% (10/12) of the patients by cranial magnetic resonance imaging and in 58.3% (7/12) by computerized tomography. Cerebrospinal fluid (CSF) examination revealed low glucose levels in 17.6% (3/17), high protein levels in 47% (8/17) and increased white blood cells with a predominance of lymphocytes in 41.2% (7/17) of the cases. CSF findings were within normal limits in 23.5% (4/17) of the patients. Empirical acyclovir treatment was given to all patients. One patient died at the acute phase of the infection while all the other 16 recovered. Since none of the CSF samples yielded bacterial growth, all of the patients were diagnosed as viral encephalitis. However, no investigation was performed to identify the viral etiology and this was the major limitation owing to the inadequacy of laboratory facilities during the study period and/or unawareness of the physicians about viral identification methods.en_US
dc.identifier.endpage320en_US
dc.identifier.issn0374-9096
dc.identifier.issue2en_US
dc.identifier.pmid18697429en_US
dc.identifier.scopus2-s2.0-52349089025en_US
dc.identifier.scopusqualityQ4en_US
dc.identifier.startpage315en_US
dc.identifier.urihttps://hdl.handle.net/20.500.14551/20798
dc.identifier.volume42en_US
dc.identifier.wosWOS:000257024300013en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isotren_US
dc.publisherAnkara Microbiology Socen_US
dc.relation.ispartofMikrobiyoloji Bultenien_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectEncephalitisen_US
dc.subjectClinical Evaluationen_US
dc.subjectDiagnosisen_US
dc.subjectHerpes-Simplex Encephalitisen_US
dc.subjectCentral-Nervous-Systemen_US
dc.subjectDiagnosisen_US
dc.subjectInfectionsen_US
dc.titleRetrospective evaluation of the cases prediagnosed as viral encephalitis in Trace University Hospital between the period of 2000-2005en_US
dc.typeArticleen_US

Dosyalar