Diaphragmatic movements in ankylosing spondylitis patients and their association with clinical factors: an ultrasonographic study

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dc.authorwosidCakir, Necati/AAG-7283-2019
dc.authorwosiderer, burak/AAR-5866-2021
dc.contributor.authorUnlu, Ercument
dc.contributor.authorPamuk, Omer Nuri
dc.contributor.authorErer, Burak
dc.contributor.authorDonmez, Salim
dc.contributor.authorCakir, Necati
dc.date.accessioned2024-06-12T10:51:46Z
dc.date.available2024-06-12T10:51:46Z
dc.date.issued2012
dc.departmentTrakya Üniversitesien_US
dc.description.abstractWe compared diaphragmatic motion between ankylosing spondylitis (AS) patients and controls, as assessed by the ultrasonographic method. We included 33 consecutive AS patients (19 males, 14 females) followed up at our center and 14 apparently healthy controls (8 males, 6 females) into our study. AS patients fulfilled the modified New York classification criteria for AS. Patients' demographic and clinical data, functional parameters, and radiographic findings were recorded down. By evaluating the motion of right and left diaphragm during deep expirium and inspirium, the mean diaphragmatic motion was determined by ultrasonography. Diaphragmatic motion in AS patients was less than in controls, but the difference was not significant (68.9 +/- A 17 mm vs. 77.8 +/- A 22.4 mm, P = 0.14). Diaphragmatic motion in AS patients who were active according to BASDAI score (> 4) was not different from inactive patients (70.4 +/- A 20.5 vs. 67.5 +/- A 13.5, P > 0.05). The mean diaphragmatic motion had a positive correlation with occiput-to-wall distance (r = 0.35, P = 0.048); and negative correlations with cervical rotation (r = -0.45, P = 0.01) and modified Schober test (r = -0.34, P = 0.05) in AS patients. We did not detect any association of mean diaphragmatic motion with thoracic expansion on deep expiration. Diaphragmatic motion in AS does not differ significantly from the control group. Factors like disease activation, chest expansion, and the severity of radiographic findings do not affect diaphragmatic motion. There is no compensatory increase in diaphragmatic motion in AS.en_US
dc.identifier.doi10.1007/s00296-010-1657-1
dc.identifier.endpage437en_US
dc.identifier.issn0172-8172
dc.identifier.issue2en_US
dc.identifier.pmid21120494en_US
dc.identifier.scopus2-s2.0-84857043534en_US
dc.identifier.scopusqualityQ2en_US
dc.identifier.startpage435en_US
dc.identifier.urihttps://doi.org/10.1007/s00296-010-1657-1
dc.identifier.urihttps://hdl.handle.net/20.500.14551/18480
dc.identifier.volume32en_US
dc.identifier.wosWOS:000299507900024en_US
dc.identifier.wosqualityQ3en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherSpringer Heidelbergen_US
dc.relation.ispartofRheumatology Internationalen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectAnkylosing Spondylitisen_US
dc.subjectDiaphragmatic Motionen_US
dc.subjectUltrasonographyen_US
dc.subjectPulmonary-Functionen_US
dc.titleDiaphragmatic movements in ankylosing spondylitis patients and their association with clinical factors: an ultrasonographic studyen_US
dc.typeArticleen_US

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