Etiology of neonatal gastric perforations

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dc.authorwosidDuran, Rıdvan/C-1065-2015
dc.contributor.authorDuran, Ridvan
dc.contributor.authorInan, Mustafa
dc.contributor.authorVatansever, Uelfet
dc.contributor.authorAladag, Nuekhet
dc.contributor.authorAcunas, Betuel
dc.date.accessioned2024-06-12T10:56:21Z
dc.date.available2024-06-12T10:56:21Z
dc.date.issued2007
dc.departmentTrakya Üniversitesien_US
dc.description.abstractBackground: Neonatal gastric perforation (NGP) is a rare event and its etiology is still controversial. Although it has previously been described as spontaneous, recently some risk factors have been reported to be associated with the development of NGP including prematurity and nasal ventilation. The purpose of the present paper was to report and discuss etiology, clinical features, and outcome of the authors' NGP cases over a 10 year period. Methods: Charts of five infants with NGP was reviewed in terms of gender, birthweight, gestational age, time of diagnosis, associated disease, site of perforation, type of surgery performed, and clinical outcome. Results: There were three boys and two girls with a mean birthweight and gestational age of 1650 g and 32 weeks, respectively. Three of them were premature. Mean perforation time was day 10 postnatally. Three infants had associated problems including prematurity, respiratory distress syndrome type 1, necrotizing enterocolitis, mechanical ventilator support, and one of them had tracheaesophageal fistula. Mothers of two out of these three infants had chorioamnionitis. One full-term infant received dexamethasone because of brain edema. Only one patient had no associated problem. Perforation occurred in the lesser curvature in three infants and in the greater curvature in two infants. Mortality rate was 60%. Conclusions: Contrary to previous literature, and similar to recent publications, it was found that essentially low-birthweight infants with tracheaesophageal fistula or chorioamnionitis and full-term babies on steroid therapy may have a risk for NGP, suggesting that an infant with contributing factors should be monitored more carefully for the development of NGP.en_US
dc.identifier.doi10.1111/j.1442-200X.2007.02427.x
dc.identifier.endpage630en_US
dc.identifier.issn1328-8067
dc.identifier.issn1442-200X
dc.identifier.issue5en_US
dc.identifier.pmid17875089en_US
dc.identifier.scopus2-s2.0-34548682563en_US
dc.identifier.scopusqualityQ3en_US
dc.identifier.startpage626en_US
dc.identifier.urihttps://doi.org/10.1111/j.1442-200X.2007.02427.x
dc.identifier.urihttps://hdl.handle.net/20.500.14551/19768
dc.identifier.volume49en_US
dc.identifier.wosWOS:000249493400016en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.language.isoenen_US
dc.publisherWileyen_US
dc.relation.ispartofPediatrics Internationalen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectChorioamnionitisen_US
dc.subjectNeonatal Gastric Perforationen_US
dc.subjectPrematurityen_US
dc.subjectTracheaesophageal Fistulaen_US
dc.subjectGastrointestinal Perforationen_US
dc.titleEtiology of neonatal gastric perforationsen_US
dc.typeArticleen_US

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