A case of Guillain-Barre syndrome in a patient with small cell lung cancer treated with chemotherapy

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dc.authoridHacıbekiroğlu, İlhan/0000-0002-0333-7405
dc.authoridCicin, Irfan/0000-0002-7584-3868;
dc.authorwosidHacıbekiroğlu, İlhan/JCN-7264-2023
dc.authorwosidCicin, Irfan/AAQ-5575-2020
dc.authorwosidErdogan, Bulent/AAA-9781-2021
dc.contributor.authorTurkmen, Esma
dc.contributor.authorErdogan, Bulent
dc.contributor.authorHacibekiroglu, Ilhan
dc.contributor.authorKodaz, Hilmi
dc.contributor.authorUzunoglu, Sernaz
dc.contributor.authorCelik, Yahya
dc.contributor.authorCicin, Irfan
dc.date.accessioned2024-06-12T11:02:51Z
dc.date.available2024-06-12T11:02:51Z
dc.date.issued2014
dc.departmentTrakya Üniversitesien_US
dc.description.abstractGuillain-Barre syndrome (GBS) is an acute inflammatory demyelinating polyradiculoneuropathy characterized by bilateral progressive symmetrical paralysis. GBS is rarely seen neuropathy in cancer patients. In the literature some cases of GBS associated with anticancer chemotherapy. In this case; the guillain-barre syndrome developed after the treatment of a 59-year-old male patient with metastatic small cell lung carcinoma who admitted to hospital with neutropenic fever after cisplatin/etoposide chemoteraphy regime is presented. The patients complained of bilteral progressive symmetrical paralysis in upper and lower limbs with depressed deep tendon reflexes and hypoesthesia. There was no pathological findings on electromyography. There was no a sign at radiological imaging that explained cranial and spinal mestastasis. The cerebrospinal fluid had albuminocytologic dissociation. Decline in tumoral lesions were detected on chest radiography. Accompanied by clinical and laboratory findings, a diagnosis of Guillain-Barre syndrome was considered. Semptoms completely disapperared after intravenous immunoglobulin for five days. Recurrence did not during follow-up. The patients was administered a total of 4 cycles of cisplatin/etoposide chemotherapy. The patient died due to disease progression six months later. We think that, in this case GBS was not a paraneoplatic syndrome because there was more than 50% tumor shrinkage. We propose GBS was induced by infection and chemotherapy rather than malignancy.en_US
dc.identifier.doi10.5505/tjoncol.2014.1126
dc.identifier.endpage107en_US
dc.identifier.issn1300-7467
dc.identifier.issue3en_US
dc.identifier.scopus2-s2.0-84911941909en_US
dc.identifier.scopusqualityQ4en_US
dc.identifier.startpage104en_US
dc.identifier.urihttps://doi.org/10.5505/tjoncol.2014.1126
dc.identifier.urihttps://hdl.handle.net/20.500.14551/21446
dc.identifier.volume29en_US
dc.identifier.wosWOS:000439268000004en_US
dc.identifier.wosqualityN/Aen_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.language.isotren_US
dc.publisherKare Publen_US
dc.relation.ispartofTurk Onkoloji Dergisi-Turkish Journal Of Oncologyen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectGuillain-Barre Sydromeen_US
dc.subjectSmall Cell Lung Canceren_US
dc.subjectNeuropathyen_US
dc.titleA case of Guillain-Barre syndrome in a patient with small cell lung cancer treated with chemotherapyen_US
dc.typeArticleen_US

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