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Öğe Arterial distensibility as determined by carotid-femoral pulse wave velocity in patients with Behcet's disease(Springer, 2005) Kürüm, T; Yildiz, M; Soy, M; Özbay, G; Alimgil, L; Tüzün, BBehcet's disease (BD) is a chronic, multisystem disorder characterized by genital and oral aphthae, skin lesions, uveitis, and tendency to thrombosis. Pulse wave velocity (PWV) is an important factor in determining cardiovascular mortality and morbidity. It is an index of arterial wall stiffness and inversely related to the arterial distensibility. In this study we investigated the arterial distensibility in BD by PWV. We studied 14 patients with BD ( 18 - 44 years old, 10 men) and 28 healthy subjects ( 18 - 39 years old, 21 men) without known cardiovascular disease. Arterial distensibility was assessed by automatic carotid-femoral PWV measurement using the Complior Colson device. PWV is calculated from measurements of pulse transit time and the distance traveled by the pulse between two recording sites, according to the following formula: pulse wave velocity (m/s)= distance (m)/transit time(s). The mean ages, systolic blood pressure, diastolic blood pressure, pulse pressure, heart rate, and PWV of Behcet's disease and control subjects were 32.1 +/- 7.4 vs 27.9 +/- 6.1 years, 112.9 +/- 12.0 vs 108.7 +/- 10.0 mmHg, 72.1 +/- 10.7 vs 67.7 +/- 7.5 mmHg, 40.7 +/- 12.2 vs 41.0 +/- 10.7 mmHg, 74.1 +/- 10.2 vs 77.2 +/- 10.1 bpm, and 8.4 +/- 1.4 vs 8.5 +/- 1.1 m/s, respectively. Differences between all parameters studied were not found to be statistically significant ( p> 0.05). The carotid-femoral PWV, an index of arterial stiffness and a marker of atherosclerosis, is not increased in patients with BD compared with control subjects.Öğe A case of adult-onset Still's disease presenting with angioedema(Springer-Verlag, 2004) Soy, MAdult-onset Still's disease (AOSD) is the adult form of Still's disease. In addition to classic symptoms, rarely urticaria may be seen. The author here presents a case of AOSD in which the patient presented with angioedema and urticaria.Öğe A case of primary anti phospholipid syndrome who developed acute myocardial infarction followed by early-onset pre-eclampsia(Springer-Verlag, 2003) Kürüm, T; Soy, M; Karahasanoglu, E; Özbay, G; Sayin, NC[Abstract Not Available]Öğe Increased pulse wave velocity and shortened pulse wave propagation time in young patients with rheumatoid arthritis(Elsevier Science Inc, 2004) Yildiz, M; Soy, M; Kurum, T; Ozbay, GBACKGROUND: Rheumatoid arthritis (RA) is a systemic immune and inflammatory disease associated with excess cardiovascular morbidity and mortality. Pulse wave velocity (PWV) is an index of arterial stiffness and a marker of cardiovascular events. OBJECTIVE: To investigate arterial stiffness using carotid-femoral (aortic) PWV measurements in young patients with RA. PATIENTS AND METHODS: Eight patients (aged 21 to 34 years, seven women, mean RA duration 13.8+/-12.6 months) with RA according to the criteria of the American College of Rheumatology, and eight age- and sex-matched control subjects (aged 22 to 34 years, seven women) were recruited. Aortic PWV was determined using an automatic device, the Complior (Complior Colson, France), which allowed on-tine pulse wave recording and automatic calculation of PWV RESULTS: The carotid-femoral PWV, systolic blood pressure and heart rate were higher in young patients with RA than in sex- and age-matched control subjects (P=0.03, P=0.02 and P=0.002, respectively). In the young patients with RA, pulse wave propagation time between measurement sites was significantly shorter than in the control group (P=0.02). There were no significant differences in the sex, age, body mass index, waist to hip ratio, diastolic blood pressure, mean blood pressure or pulse pressure between the two groups (P=1.00, P=0.71, P=0.20, P=0.66, P=0.55, P=0.07 and P=0.11, respectively). CONCLUSION: The carotid-femoral PWV is increased and Pulse wave propagation time is decreased in young patients with RA. Measurements of carotid-femoral PWV may provide a simple and noninvasive technique for identifying patients at increased risk of vascular disease.Öğe Lymphadenopathy in adult-onset Still's disease mimicking peripheral T-cell lymphoma(Springer-Verlag, 2004) Soy, M; Ergin, M; Paydas, SLymphadenopathy (LAP) that is seen in adult onset Still's disease (AOSD) may be confused with lymphoma. Here we present a patient with AOSD and with LAP that histopathologically mimicked T-cell lymphoma.Öğe Mitral valve vegetation and cerebral emboli in a primary antiphospholipid syndrome patient who had hepatitis C virus infection(Springer-Verlag, 2003) Pamuk, ÖN; Çakir, N; Soy, M; Aktoz, M; Çelik, Y; Akdemir, OWe report the case of 36-year-old woman who came to us with a history of recurrent miscarriages and who was later diagnosed as having primary antiphospholipid syndrome (PAPS) and chronic hepatitis C virus (HCV) infection. The patient was referred to us with generalised seizures; cranial MRI revealed multiple embolic infarcts in both frontal lobes and a focal cortical infarct in the left frontoparietal lobe. Her echocardiography showed mitral valve vegetation and insufficiency. The patient was put on oral anticoagulant therapy and during her 8-month follow-up period no thrombotic events occurred. We report this case because it was the first in which PAPS, valvular disease, a cerebral embolic event and HCV infection were coexistent in the same patient. We also review other cases in which there was valvular vegetation and a cerebral ischaemic event associated with PAPS.Öğe Nocardiosis in a patient with primary anti-phospholipid syndrome(Springer Heidelberg, 2006) Soy, M; Oktun, MT; Tunçbilek, N; Ermantas, N; Ökten, Ö; Altinay, G; Turgut, BAntiphospholipid syndrome is an autoimmune disease that is characterised by tendency to thrombosis, obstetrical and hematological complications. Corticosteroids may be useful for therapy of some features of this syndrome, such as thrombocytopenia. Nocardia is an important opportunistic infectious agent in immunocompromised hosts, i.e. in patients taking corticosteroids. It is important to be aware of these rare complications, which are correlated with the prognosis. In this paper, we report a patient with primary antiphospholipid syndrome treated by corticosteroid, who developed disseminated nocardiosis.Öğe A primary Sjogren's syndrome patient with distal renal tubular acidosis, who presented with symptoms of hypokalemic periodic paralysis(Springer Heidelberg, 2005) Soy, M; Pamuk, ÖN; Gerenli, M; Çelik, YAlthough renal tubular acidosis (RTA), secondary to autoimmune interstitial nephritis, develops in a large proportion of patients with Sjogren's syndrome (SS), most of the subjects are asymptomatic. Here, we shall present a 39-year-old female patient who came to us with hypokalemic periodic paralysis (HPP), and who was later diagnosed with distal RTA. The patient, who had xerostomia and xerophthalmia for a long period of time, was diagnosed with primary SS from serologic and histologic findings. The patient recovered by being prescribed potassium replacement therapy. Although renal biopsy was not performed, corticosteroids were administered because HPP indicated severe interstitial nephritis. HPP did not reoccur during a 2-year follow-up period. We also review cases with SS-related distal RTA and HPP.Öğe Splenic infarction in a pregnant woman with systemic lupus erythematosus(Springer London Ltd, 2005) Soy, M; Sayin, NC; Unlü, EHere we describe a 20-year-old pregnant woman with systemic lupus erythematosus who had high anticardiolipin antibodies and presented with splenic infarction.Öğe Thrombosis in systemic lupus erythematosus(Springer-Verlag, 2003) Pamuk, GE; Turgut, B; Vural, Ö; Demir, M; Soy, M; Bozkurt, G; Çelik, HThrombosis in the venous or arterial system is quite common in systemic lupus erythematosus (SLE). We describe a young female patient whose first presentation was in the form of deep venous thrombosis of the right lower extremity. Her family history for thrombosis was positive and further studies revealed her to have SLE. Genetic studies showed that she had thrombophilic mutations of factor V, prothrombin and methylene tetrahydrofolate reductase genes. Her therapeutic response to anticoagulant therapy was satisfactory. The presence of inherited thrombophilic mutations must be searched for in SLE patients with thrombosis, especially in cases with a positive family history.Öğe Vasculitis and cerebral infarcts(B M J Publishing Group, 2003) Soy, M; Gerenli, M; Çelik, Y[Abstract Not Available]
