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Öğe Alcohol intake and sexually transmitted diseases(Elsevier Science Inc, 1999) Tüzün, B; Tüzün, Y; Wolf, R[Abstract Not Available]Öğe Anogenital lesions (viral diseases and ectoparasitic infestations)(Elsevier Science Inc, 2002) Tüzün, B; Saygin, A; Wolf, R; Özdemir, M; Tüzün, Y[Abstract Not Available]Öğe Comparison of two surgical approaches for treating vitiligo(Blackwell Publishing Ltd, 2002) Özdemir, M; Çetinkale, O; Wolf, R; Kotogyan, A; Mat, C; Tüzün, B; Tüzün, YBackground Surgical techniques have recently been introduced for patients with vitiligo. Randomized controlled trials have not been performed. Aim To compare the efficacy and side-effects of two surgical methods (suction blister vs. thin split-thickness graft technique) for the treatment of vitillgo. Methods Three suction blisters, approximately 0.8 cm in diameter, made with a special tool attached to a vacuum extractor, which were converted to erosions by removal of the roofs of the bullae, and one round erosion of approximately the same diameter, made using a silver knife, were created. One roof of a blister and two cutaneous thin split-thickness grafts taken from the gluteal region were transferred onto recipient vitiliginous areas; one was left alone. The results were evaluated by one non-blind and two blind observers bimonthly during the 3-month follow-up period. Results Repigmentation rates were 25-65% in the suction blister technique and 90% in the thin split-thickness graft technique (P < 0.001). Conclusions The thin split-thickness graft technique is superior to the suction blister technique in treating vitiligo.Öğe Familial erythromelanosis follicularis and chromosomal instability(European Acad Dermatology & Venereology, 2001) Tüzün, Y; Wolf, R; Tüzün, B; Özdemir, M; Demirkesen, C; Deviren, A; Kotogyan, AWe report a 17-year-old male patient with erythromelanosis follicularis faciei et colli (EFFC), oral leucokeratosis and diabetes mellitus without islet cell antibody. His sister also had minimal findings of EFFC and minimal follicular papules on her shoulders and extensor surfaces of the arms. The father had only fine follicular papules, but no erythromelanosis. Skin and mucous membrane lesions of the proband were investigated histopathologically. Interestingly, in peripheral lymphocyte cultures of the family members, chromosomal breakage was not observed spontaneously, but it was seen with nitrogen mustard, although this disease may be of autosomal recessive inheritance. Thus, we suggest that EFFC may be a poly-aetiological disorder (i.e. familial and environmental) and might be considered one of the chromosomal instability syndromes.Öğe Increased basic fibroblast growth factor levels in serum and blister fluid from patients with vitiligo(Taylor & Francis As, 2000) Özdemir, M; Yillar, G; Wolf, R; Yillar, O; Ünal, G; Tüzün, B; Tüzün, YBasic fibroblast growth factor (bFGF) is a pleiotropic growth factor which has a high capacity for stimulating normal melanocyte proliferation and suppressing melanogenesis, The close and complicated relationship between bFGF, melanocyte proliferation and melanogenesis raises the theoretical possibility that bFGF may also be involved in the pathomechanism leading to vitiligo, The aim of this study was to compare the serum and suction blister fluid bFGF levels of vitiligo patients (9 females, 11 males) with those of healthy controls (3 females, 8 males). Vitiliginous skin-blister fluid bFGF levels and serum levels were significantly higher in vitiligo patients compared with healthy normal controls. Our data indicate that bFGF might be involved in the pathogenetic chain of events leading to vitiligo. Further studies are needed to define the exact role of bFGF and various other melanocytic mitogens in this disease.Öğe Miscellaneous treatments, I(Elsevier Science Inc, 2002) Wolf, R; Matz, H; Orion, E; Tüzün, B; Tüzün, Y[Abstract Not Available]Öğe Miscellaneous treatments, II(Elsevier Science Inc, 2002) Wolf, R; Orion, E; Matz, H; Tüzün, Y; Tüzün, B[Abstract Not Available]Öğe Öğe Oral therapeutics and oral cosmetics(Elsevier Science Inc, 2001) Tüzün, B; Firatli, S; Tüzün, Y; Firatli, E; Wolf, R[Abstract Not Available]Öğe The plight of descriptive dermatology and clinical investigation in an evidence-based world(Mosby, Inc, 2002) Wolf, R; Tüzün, B; Tüzün, Y[Abstract Not Available]Öğe Recurrent aphthous stomatitis and smoking(Blackwell Science Ltd, 2000) Tüzün, B; Wolf, R; Tüzün, Y; Serdaroglu, SBackground Several studies have reported cigarette smoking to have a beneficial protective effect on recurrent aphthous stomatitis (RAS). In this study, we evaluated once again the incidence of smoking in RAS patients compared with controls. This study differs from most previous ones in that the patients were diagnosed by direct observation of active lesions by a dermatologist. Methods Thirty-four patients with RAS who were seen at the dermatology clinic during a period of 2 years were compared with 115 outpatients with other skin diseases and 20 healthy hospital personnel who had no history of aphthae, with regard to their smoking habits. Results Among the 34 patients with RAS, 8.8% were active smokers compared with a significantly higher percentage (25.2%) among the control subjects. Conclusions In agreement with others, we found a negative epidemiologic association between smoking and RAS. This finding can be used to clarify the cause and pathogenesis of the disease, and possibly to identify better treatment or preventive options than those currently available.Öğe The relationship between Helicobacter pylori IgG antibody and autologous serum test in chronic urticaria(Blackwell Science Ltd, 2000) Hizal, M; Tüzün, B; Wolf, R; Tüzün, YBackground In chronic urticaria, suspicious factors are Helicobacter pylori (HP) infection and autoimmunity, as well as other etiologic agents. Autologous serum injection is the first step to identify autoimmune urticaria. Our study was performed to determine the prevalence of HP infection in patients with chronic urticaria, and to evaluate the results of autologous serum testing in chronic urticaria patients who had HP antibodies. Methods HP immunoglobulin G (IgG) antibody was defined by an immunochromatographic method in 61 chronic urticaria patients and 15 healthy persons. Autologous serum testing was performed in 32 chronic urticaria patients. Results HP IgG antibody was found to be positive in 41% of chronic urticaria patients and 26% of healthy controls (chi(2) = 7.82, P = 0.005). Autologous serum testing was positive in 41% of chronic urticaria patients who had HP IgG antibody. This ratio was 14.3% in chronic urticaria patients who did not have HP IgG antibody (chi(2) = 9.23, P = 0.002). Conclusions In this study, autologous serum testing was found to highly positive in chronic urticaria patients with HP IgG antibody, but the relationship between autoimmunity and HP infection requires further investigations.
